319) All the protein initiation codons are ATG, except |
320) All the protein initiation codons are ATG, except |
321) At the protein level, ADNP co-immunoprecipitated |
322) ecrease of APAF1, Beclin1 and PAK6 at the protein level, proving the regulating infl |
323) of the highest growth rates within animal protein production during recent decades a |
324) e expression of antigens leading to viral protein production in the upper layers of |
325) Phylogenetic analysis of protein sequences revealed a close related |
326) japonica protein-coding sequences start with a typi |
327) higher kD values are, the more stable the protein solutions are, as long as the ther |
328) Concerning colloidal stability, protein solutions of different kD values a |
329) Comparison between protein structures and multiple sequence a |
330) Comparison of normal and mutant protein structures of Nsun7 indicated that |
331) at interacts with FtsZ is MinC, a 25 kDa protein that has two domains. |
332) ize the current knowledge on pneumococcal proteins that are being investigated as po |
333) Total DNA, collagen and sGAG protein were also greater on POSS-PCU scaf |
334) PSPN mRNA and protein were significantly (P < 0.0 |
335) t with activity-dependent neuroprotective protein (ADNP) and ADNP2, which we have pr |
336) ocessing of endothelial amyloid precursor protein (APP). |
337) s from the Activation-associated Secreted Protein (ASP) family were targets for knoc |
338) ion in the gene encoding bone morphogenic protein (BMP) type II receptor (ACVR1/ALK2 |
339) aggrecan and cartilage oligomeric matrix protein (COMP), were assessed at both the |
340) nd phospho- cAMP response element binding protein (CREB) expression in a cyclic AMP- |
341) sure to Al fumes and levels of C-reactive protein (CRP) and alpha-1-antitrypsin (A1A |
342) evels of interleukin 6 (IL-6), C-reactive protein (CRP), soluble intercellular adhes |
343) s measured, and serum ADMA and C-reactive protein (CRP; a marker of inflammation) le |
344) e that express enhanced green fluorescent protein (EGFP) under the control of the ty |
345) om a loss of Fragile X mental retardation protein (FMRP). |
346) y associated with glial fibrillary acidic protein (GFAP(+)) astrocyte processes that |
347) ayed to determine glial fibrillary acidic protein (GFAP) and tumor necrosis factor a |
348) ve either IT BM-derived green fluorescent protein (GFP)(+) c-kit(-) cells (PL) or BM |
349) m 3D reconstructions of green fluorescent protein (GFP)-labeled cells showed that sp |
350) rom the bone marrow of green fluorescence protein (GFP)-transgenic Sprague-Dawley (S |
351) owth arrest and DNA damage-inducible 45α protein (Gadd45α) has been characterized |
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