288) imized using the central composite design-response surface methodology with drug loa |
289) inoculum density, incubation density) by response surface methodology (RSM) led to |
290) n of ultrasonication were optimized using response surface methodology (RSM) to maxi |
291) lysaccharide gel of oleoyl alginate using response surface methodology as well as to |
292) eoyl alginate material was obtained using response surface methodology in terms of t |
293) Central composite design with response surface methodology was used to a |
294) ization of PD drug interactions different response surface models are discussed. |
295) Dose-response data suggested about the same ran |
296) ar models (GLM), so that it can deal with response data generated from a variety of |
297) hodology is illustrated with several item response data sets, and it is shown that t |
298) Moreover, it permits predictor and/or response data to be functional. |
299) g the relationships between predictor and response data. |
300) nent accounts for the maximum variance of response data. |
301) Non-response is a serious threat to the extern |
302) Myc transcriptional activity and clinical response is favorable. |
303) Although the innate response is largely beneficial, it also co |
304) on to a rapid onset, a fast offset of the response is required. |
305) ild and (b) clarify that the most ethical response is to simply not include that chi |
306) th, tumorigenesis, cell invasion and drug response, is frequently activated in numer |
307) ve percent of dogs experienced a complete response for a median of 81 days (range 42 |
308) y, with apixaban also showing a favorable response for major bleeding and total disc |
309) initial data obtained on the therapeutic response for refractory CW metastasis or r |
310) s by caries experience and by global item response for the respective age-groups, wi |
311) nd associations with three common sensory response patterns (hyperresponsiveness; hy |
312) nimize the distance between observed item response patterns and ideal response patte |
313) Critically, response patterns in these regions were in |
314) These models recognize that response patterns tend to deviate from the |
315) ls and time-course of induction of stress response proteins and apoptosis-related pr |
316) nificant increase in the levels of stress response proteins and apoptotis- related p |
317) We found that the acute-phase response proteins serum amyloid A (SAA) 1 |
318) fiers, signaling molecules and DNA damage response proteins. |
319) eb-based physician leader survey improved response rates but not response quality or |
320) Adding postal follow-up increased response rates by 20-25 percentage points; |
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