283) predicting bone structural remodelling in cancer are also briefly discussed. |
284) The hallmarks of cancer are described as the distinctive an |
285) n tumorigenesis and genetic complexity in cancer are discussed. |
286) s screening and early detection of breast cancer are important in reducing cancer mo |
287) long-regarded risk factors for esophageal cancer are tobacco, alcohol, and reflux es |
288) epidemiologic complexities of esophageal cancer are vast, rendering screening and p |
289) source of agents for NFκB inhibition in cancer therapy. |
290) echanisms that favor their application in cancer therapy. |
291) c miRNAs as potential tools or targets in cancer therapy. |
292) y of life as a consequence of the primary cancer therapy. |
293) expectations for possible applications in cancer therapy. |
294) possible mechanisms for its role in liver cancer therapy. |
295) gnificantly decreased or silenced in lung cancer tissues and cell lines associated w |
296) in TAMs was analyzed in human pancreatic cancer tissues and in a genetic mouse mode |
297) hylation in 52 of 98 (53.1%) primary lung cancer tissues and in nine of 10 (90%) cel |
298) ein expression was performed using breast cancer tissues from the proband with doubl |
299) Therefore, we analyzed mouse skin cancer tissues induced by the 2-stage carc |
300) from A2780 cell line and primary ovarian cancer tissues. |
301) ence of TRPV1 in human and canine mammary cancer cells, MCF-7 and CF.41, respectivel |
302) lates Dishevelled (DVL) protein levels in cancer cells, and DVL along with TIAM1 are |
303) , was recently shown to be transcribed in cancer cells, but its clinical relevance a |
304) In high-invasive breast cancer cells, miR-106b is upregulated by T |
305) of fibroblastic TGF-β pathway on breast cancer cells, we used cell contact-depende |
306) C diet increases human ER-positive breast cancer progression and modulates natural k |
307) nificance and biological role of AKIP1 in cancer progression remain largely unknown. |
308) beneficial, resulting in reduced rates of cancer progression. |
309) rucial roles in cell cycle regulation and cancer progression. |
310) pithelial cells, a model for early breast cancer progression. |
311) the international recognised standing of cancer registries and register-based resea |
312) ogically confirmed and recorded in the TT cancer registries between 1995 and 2005, w |
313) ally significant trends in incidence with cancer registries data not only depends on |
314) set for linkages of biobanks, cohorts and cancer registries is proposed. |
315) in the linkage of cohorts and biobanks to cancer registries is required in order to |
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