280) ous findings to provide evidence for post-response selection expression of the Simon |
281) nt expression of the Simon effect in post-response selection stages. |
282) uishable from a dorsal pathway related to response selection demands. |
283) pression of a conflict that occurs at the response selection stage. |
284) t could not be fully dissociated from the response selection stage. |
285) eral components and independently affects response selection stages as well as other |
286) gether with the subsequent first phase of response selection. |
287) to a more parallel and hence inefficient response selection. |
288) imized using the central composite design-response surface methodology with drug loa |
289) inoculum density, incubation density) by response surface methodology (RSM) led to |
290) n of ultrasonication were optimized using response surface methodology (RSM) to maxi |
291) lysaccharide gel of oleoyl alginate using response surface methodology as well as to |
292) eoyl alginate material was obtained using response surface methodology in terms of t |
293) Central composite design with response surface methodology was used to a |
294) ization of PD drug interactions different response surface models are discussed. |
295) Dose-response data suggested about the same ran |
296) ar models (GLM), so that it can deal with response data generated from a variety of |
297) hodology is illustrated with several item response data sets, and it is shown that t |
298) Moreover, it permits predictor and/or response data to be functional. |
299) g the relationships between predictor and response data. |
300) nent accounts for the maximum variance of response data. |
301) Non-response is a serious threat to the extern |
302) Myc transcriptional activity and clinical response is favorable. |
303) Although the innate response is largely beneficial, it also co |
304) on to a rapid onset, a fast offset of the response is required. |
305) ild and (b) clarify that the most ethical response is to simply not include that chi |
306) th, tumorigenesis, cell invasion and drug response, is frequently activated in numer |
307) ve percent of dogs experienced a complete response for a median of 81 days (range 42 |
308) y, with apixaban also showing a favorable response for major bleeding and total disc |
309) initial data obtained on the therapeutic response for refractory CW metastasis or r |
310) s by caries experience and by global item response for the respective age-groups, wi |
311) nd associations with three common sensory response patterns (hyperresponsiveness; hy |
312) nimize the distance between observed item response patterns and ideal response patte |
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