276) Pathogenic mutations in the KCNQ1 gene were detected in all JLNS patients. |
277) Eight tRNA genes and one protein-coding gene were encoded on light strand, the oth |
278) en the A + T-rich region and the nad2 gene were failed to sequence because of th |
279) ction since all substitutions within this gene were non-synonymous. |
280) the transforming growth factor (TGF) β3 gene were selected as transgenic companion |
281) entration of 0.1 μM, Pb(2+) induced IL-8 gene activation in gastric carcinoma AGS c |
282) l, we integrate negative regulations into gene activation process, and make the conv |
283) gnal transduction of Pb(2+) -induced IL-8 gene activation. |
284) cessary component of Pb(2+) -induced IL-8 gene activation. |
285) Gene delivery takes advantage of cellular |
286) ture strategy in conjunction with TGF-ß3 gene delivery could be a promising approac |
287) Two typical viral vectors for gene delivery include lentiviral vector fo |
288) se mechanism and explore these factors in gene delivery. |
289) c Ewing sarcoma, an insertion of the MIC2 gene encoding CD99 from Xp to 10p and a su |
290) AP3K-related serine-threonine kinase (the gene encoding COT/Tpl2) as a kinase respon |
291) om a spontaneous missense mutation in the gene encoding bone morphogenic protein (BM |
292) tion is an expanded CAG repeat in the HTT gene encoding the Huntingtin protein, and |
293) g/kg) increased TH and μ-opioid receptor gene expressions in CB2 KO mice, whereas t |
294) e hydroxylase (TH) and μ-opioid receptor gene expressions were also measured in the |
295) The highest levels of gene expressions were in the 5-min group a |
296) in reaction (RT-PCR) to confirm stem cell gene expressions. |
297) in-coding genes (cytb, cox1 and cox3), 12 gene fragments for the large subunit (LSU) |
298) nalysis of partial sequences of these two gene fragments from 171 individuals indica |
299) e presence of polymorphisms in all tested gene fragments in individual tissues of do |
300) the species level by sequencing 16S rRNA gene fragments. |
301) the manganese superoxide dismutase (SOD2) gene is a risk factor for primary angle cl |
302) In humans, this gene is located on chromosome 4 with 12 ex |
303) The results showed that the 12S rRNA gene is more variable than CO1. |
304) inding suggests that the embryonic STAT5A gene is primarily activated by maternal ge |
305) 0 and increased H3K27me3, contributing to gene silencing in bladder cancer cells. |
306) ges such as microRNAs (miRs)/Ago2-induced gene silencing represent complex molecular |
307) ional machinery and resulting in a robust gene silencing. |
308) cations involved in DNA methylation-based gene silencing. |
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