135) e involvement and relevance of its sialic acid binding activity is still controversi |
136) lets exhibiting significantly lower fatty acid binding protein 1 (FABP1) expression |
137) ated that oncogenic genes including fatty acid binding protein 4 (FABP4) and fibrobl |
138) e site, harbors two additional polysialic acid binding sites. |
139) Amino acid derivatives are propagated as being e |
140) olecular weight and more drug-like sialic acid derivatives binding with low nanomola |
141) dominant conformations of these two amino acid derivatives under different pHs. |
142) chelating agents of 3,4-dihydroxybenzoic acid derivatives. |
143) d by immersion into concentrated sulfuric acid for 10 min. |
144) pecimens were etched by 9.6% hydrofluoric acid for 4 min. |
145) erent phenotypes were incubated with uric acid for 48 h, respectively. |
146) n, 3-isobutyl-1-methylxanthine, and oleic acid for 8 days. |
147) sponges saturated with aqueous tranexamic acid solution and compressed onto the blee |
148) ilability of T-OA microemulsion and oleic acid solution were checked by using rat mo |
149) rve (AUC) of T-OA microemulsion and oleic acid solution were significantly enhanced. |
150) cetonitrile and 60% (v/v) orthophosphoric acid solution with flow rate of 1.5 mL/min |
151) SBS was found after additional phosphoric acid treatment in dentin groups (p < |
152) Phosphoric acid treatment seems the most promising su |
153) Implants with surface acid treatment undergo greater superficial |
154) m NaCl, distilled water) and peroxyacetic acid treatment. |
155) fficacy with coadministration of ascorbic acid (AA) and Pleurotus florida lectin (PF |
156) med to investigate the effect of ascorbic acid (AA) in reducing hemato-biochemical c |
157) Ascorbic acid (AA) is a naturally occurring phenoli |
158) -1) diet and n-3 PUFA was alpha linolenic acid (ALA) 1.68, eicosapentaenoic acid 5.6 |
159) f the population get only alpha-linolenic acid (ALA) through their diet as a source |
160) ion and the possible role of alpha lipoic acid (ALA) versus N-acetylcysteine (NAC) i |
161) Gamma-aminobutyric acid (GABA) content in hippocampal tissue |
162) romolar concentrations of γ-aminobutyric acid (GABA). |
163) ifically convert Glu into γ-aminobutyric acid (GABA). |
164) ntly affected plasma non-esterified fatty acid (NEFA) and insulin concentrations. |
165) xybutyrate (BHB) and non-esterified fatty acid (NEFA) concentrations in the blood co |
166) sorders is increased non-esterified fatty acid (NEFA) concentrations in the blood, w |
167) 1 missense mutations causing single amino acid changes (p.Arg49Trp, p.Arg58Gln, p.Pr |
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