1) Tumor cells have an increased reliance on |
2) cer drugs dosages to discriminate between tumor cells and normal cells for minimizin |
3) Differences in energy metabolism between tumor cells and normal cells offer an attr |
4) It seems that tumor cells are more sensitive to oxidized |
5) that CUX1 is highly expressed not only in tumor cells but also in TAMs. |
6) NAs were repressed in cultured Kras(G12D) tumor cells but reactivated when transplan |
7) on can also provide a growth advantage to tumor cells by regulating cellular metabol |
8) However, tumor cells can maintain a high glycolytic |
9) that lacked p53 and αv in the epithelial tumor cells exhibited high Akt activity, l |
10) ormal cells, disagreeing with report that tumor cells express exclusively PKM2. |
11) situ, to reverse the "Warburg effect" of tumor cells has been demonstrated as an ef |
12) x exhibited a higher cytotoxicity against tumor cells in vitro. |
13) ntrast, one trait shared by virtually all tumor cells is altered (dysregulated) meta |
14) NKG2D ligand expression renders tumor cells more susceptible to be killed |
15) The tumor cells possessed uniformly round nucl |
16) el, ectopic expression of SAA1 or SAA3 in tumor cells potently promoted widespread m |
17) afin confers a proliferative advantage to tumor cells through the activation of the |
18) g the MAPK and AKT pathways, which allows tumor cells to proliferate in the absence |
19) might prevent mitotic slippage and allow tumor cells to reach the threshold for apo |
20) IFs) play crucial role in the response of tumor cells to this distinct microenvironm |
21) Cx43 may influence the response of tumor cells to treatments by facilitating |
22) By immunocytochemistry, the tumor cells were positive for cytokeratins |
23) Furthermore, circulating tumor cells were significantly reduced in |
24) It is composed of tumor cells with myoepithelial differentia |
25) Immunohistochemical stainings of the tumor cells with synaptophysin, GCDFP-15, |
26) iinflammatory effects against established tumor cells. |
27) activity are likely to selectively target tumor cells. |
28) is ubiquitous and reduced in a variety of tumor cells. |
29) GLUT-1 protein and mRNA expression of the tumor cells. |
30) cell surface receptors/antigen present on tumor cells. |
31) een proposed as a better strategy to kill tumor cells. |
32) enic activity and metastatic potential of tumor cells. |
33) ere is an increased expression of MTA1 in tumor cells. |
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