1) C than CWF (77·5% versus 59·8% migrated cells). |
2) closely respected the microenvironment of cells. |
3) air mechanisms carried out by endothelial cells. |
4) ocytes that can potentially kill melanoma cells. |
5) terferes with the differentiation of K562 cells. |
6) sion and interactions of neural and other cells. |
7) onally, apoptosis occurred in PDT-treated cells. |
8) s, and focal infiltration of inflammatory cells. |
9) P < 0.05) of cell growth rate in MCF-7 cells. |
10) fferentiation potential than FCS-expanded cells. |
11) e the primary effect of NaVO(3) on CHO-K1 cells. |
12) to inhibit proliferation of human cancer cells. |
13) p-PERK, MMP-2, MMP-9, and VEGF in U-2 OS cells. |
14) and invasion of U-2 OS human osteosarcoma cells. |
15) portant biomarkers to assess apoptosis in cells. |
16) growth, differentiation and apoptosis of cells. |
17) apamycin was observed in human and canine cells. |
18) an in human leukaemia and canine lymphoma cells. |
19) ose and time-dependent signicants in A549 cells. |
20) psulomas with the resident renal capsular cells. |
21) nt way for large-scale expansion of these cells. |
22) interactions between adjacent epithelial cells. |
23) s due to the prolonged engraftment of UCB cells. |
24) within the mucosal epithelial basal layer cells. |
25) cin within the retinal pigment epithelial cells. |
26) ed by TRPV1 activation in MCF-7 and CF.41 cells. |
27) L-6) were significantly higher in treated cells. |
28) expression and protein levels in SK-Hep1 cells. |
29) e complex had an apoptotic effect on HeLa cells. |
30) n (PI3K/AKT) in human and canine melanoma cells. |
31) dium constituents, well plate plastic and cells. |
32) revealed RANKL expression by CD44+ cancer cells. |
33) ages, astrocytes and vascular endothelial cells. |
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