1) ic stability and risking the induction of cancer. |
2) of ovarian, head and neck and testicular cancer. |
3) role in carcinogenesis, such as in breast cancer. |
4) ne loss/osteoporosis in women with breast cancer. |
5) ost women undergoing treatment for breast cancer. |
6) regimen in patients with advanced breast cancer. |
7) of patients undergoing surgery for breast cancer. |
8) ntion of scientists to find a solution to cancer. |
9) en abnormal differentiation processes and cancer. |
10) h after esophagectomy to treat esophageal cancer. |
11) phography (CT-LG) in patients with breast cancer. |
12) ising vehicle for chemoprevention of skin cancer. |
13) r, and proband's son and daughter without cancer. |
14) ty and increase susceptibility to bladder cancer. |
15) ors over-expressed in non-small cell lung cancer. |
16) = 0.036, respectively) compared to breast cancer. |
17) erimental animal models and patients with cancer. |
18) cer, hepatocellular carcinoma and ovarian cancer. |
19) osition and the development of colorectal cancer. |
20) drug resistance in aggressive endometrial cancer. |
21) nflammatory bowel diseases and colorectal cancer. |
22) logical and clinical implications in lung cancer. |
23) moresistance and poor prognosis in breast cancer. |
24) protocols used for companion animals with cancer. |
25) e in the modulation and/or progression of cancer. |
26) nd believed they were at low risk of skin cancer. |
27) rbs' influence on ovarian and endometrial cancer. |
28) mise as novel anticancer agents in canine cancer. |
29) A1 haplotypes in the modulation of breast cancer. |
30) ew markers of epigenetic dysregulation in cancer. |
31) erapeutic or preventive agent for bladder cancer. |
32) had good knowledge of the dangers of skin cancer. |
33) tion contributes to disease, particularly cancer. |
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